Papel de las citocinas IL-36 en la regulación de la respuesta inmune en mucosa intestinal

Mucosal Immunology
Inflammation
Past Project
LPS/ATP stimulation induces cytokine relocalization to the plasma membrane, and conventional secretion blockade triggers no IL-36g accumulation within the cell.
Author
Affiliation

Hospital Infantil de Mexico

Published

March 10, 2024

Mucosal innate immunity functions as the rst line of defense against invading pathogens. Members of the IL-1 family are key cytokines upregulated in the in amed mucosa. In ammatory cytokines are regulated by limiting their function and availability through their activation and secretion mechanisms. IL-1 cytokines secretion is affected by the lack of a signal peptide on their sequence, which prevents them from accessing the conventional protein secretion pathway; thus, they use unconventional protein secretion pathways. Here we show in mouse macrophages that LPS/ATP stimulation induces cytokine relocalization to the plasma membrane, and conventional secretion blockade using monensin or Brefeldin A triggers no IL-36\(\gamma\) accumulation within the cell. In silico modeling indicates IL-36\(\gamma\) can pass through both the P2X7R and Gasdermin D pores, and both IL-36\(\gamma\), P2X7R and Gasdermin D mRNA are upregulated in in ammation; further, experimental blockade of these receptors’ limits IL-36\(\gamma\) release. Our results demonstrate that IL-36\(\gamma\) is secreted mainly by an unconventional pathway through membrane pores formed by P2X7R and Gasdermin D.

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